The VLED device may stop or reduce the progression of Alzheimer’s, Parkinson’s and Dementia related diseases. This occurs by maintaining a specific light frequency and amplitude to optimally activate the Microglial Cells in the brain.
The photons of light, having a specific energy level, are transported via the retina, to the occipital lobe of the brain. This energy is then discharged as flashes across the brain’s surface at the desired Gamma Wave energy level.
The photons of light, having a specific energy level, enter the eye and are converted to electrons in the eye. These electrons, having the same energy as the photon, are transported via electrochemical reactions through the retina, down the optic nerve, to the occipital lobe of the brain. This energy is then discharged as electro chemical “flashes” across the brain surface at the desired Gamma Wave energy level.
This Gamma wave energy level is required to activate the Microglial Cells. The Microglial Cells are essential for the removal of Amyloid beta protein, the primary cause of Alzheimer’s, Parkinson’s, ALS, PSP and Dementia related amyloidosis disease.
Microglia are a type of neuroglia (glial) cell located throughout the brain and spinal cord. Microglia account for 10–15% of all cells found within the brain. An adult brain contains about 100 billion nerve cells, or neurons, with branches that connect at more than 100 trillion points. Scientists call this dense, branching network a “neuron forest.”
Microglia are the resident “debris cleaning” cells. They act as the first and main form of active immune defense in the central nervous system (CNS). Microglia (and other neuroglia including astrocytes) are distributed in large non-overlapping regions throughout the CNS. Microglia are constantly scavenging the CNS for cellular debris, pathogens and Aβ plaques, damaged or unnecessary neurons and synapses, and infectious agents. Microglia must be efficient and healthy to prevent potentially fatal damage. Microglia are extremely sensitive to even slight changes in diet, medications, energy levels and pathological changes in the CNS.
Amyloid beta (Aβ or Abeta) proteins, (peptides of 36–43 amino acids), and amyloid plaques are involved in the progression of Alzheimer’s disease. Aβ molecules can aggregate to form flexible soluble oligomers, like polymers or plastic, which may exist in several forms. It is now believed that certain misfolded oligomers (known as “seeds”) can induce other Aβ molecules to also take the misfolded oligomeric form, leading to a chain reaction akin to a prion (“mad cow”) infection. The seeds or the resulting amyloid plaques are toxic to nerve cells. These plaques will accumulate on the neuron cell membrane and along the axion, slowly killing the neuron cell. The other protein implicated in Alzheimer’s disease, tau protein, also forms such prion-like misfolded oligomers, and there is some evidence that misfolded Aβ can induce tau to misfold.
We’re all forgetful at times, regardless of our age and mental health. However, as we age, our risk of dementia increases, and by the age of 85 almost 35-percent of those in your age group will be afflicted with this degenerative disorder that causes gradually and worsening memory loss and mental skills.
If a loved one shows the following ten early warning signs of dementia, book an appointment to see a doctor immediately…
Mild cognitive impairment (or as doctors refer to it, MCI) explains memory loss that is more pronounced than typical age-related forgetfulness. MCI will cause an inability to recall short term memory (or recent events) such as what you did yesterday or last week. However, suddenly a strong ability to remember specifics from the past will occur.
A study from Rutgers University, estimates that roughly 4-percent of Americans between the ages of 65 and 74-years old have Alzheimer’s Disease, with the percentage rising steady as we grow. The same research estimates that roughly 50-percent of all Americans over 85-years of age suffer with Alzheimer’s Disease and that more than half of all dementia cases are misdiagnosed and actually Alzheimer’s Disease.
Difficulty with coordination and motor functions will occur as the condition progresses and can take years, but occurs when you lose the physical ability to perform routine tasks (i.e., go to the washroom, drive to your grocery store, operate the stove) and need 24-hour care for your own safety.
In a research study, published by the U.S. National Institutes of Health, researchers found that age-related cognitive and motor decline may share a common causation, meaning that the reach of Alzheimer’s Disease may represent the “tip of the iceberg” of cognitive and motor dysfunction currently dubbed “normal aging” in older persons without Alzheimer’s Disease and Dementia.
Problems with disorientation, for example getting lost on a routine trip to the same doctor’s office you’ve been going to for years is an early sign of Alzheimer’s disease or Dementia. For instance, an individual with Alzheimer’s disease suffers death of the nerve cells very gradually, over a period of years, which causes them to struggle with thinking, decision-making and memory, which makes is confusing and scary, and often causes them to withdrawal from normal day-to-day life.
For example, this may cause a sufferer to become confused or even lost when out on their own and may not be able to remember where they are, how they got there, or how and where to return home. They may gradually start to forget familiar people, places, facts, what day it is, year it is, or what time of the day it is.
Personality changes that may include the opposite manners or personality traits—for example, becoming cranky when you were always easy going, becoming blissful when you were always a curmudgeon, or just being inappropriate in public can signify the onset of Dementia.
Many families who have a relative with Alzheimer’s almost always admit that the behavioral and psychiatric changes are the most challenging and disturbing aspect of the disease. In the early stages, Alzheimer’s sufferers commonly become anxious, depressed, and irritable due to confusion and fear. In the later stages, Alzheimer’s sufferers can suffer with paranoia and emotional distress that shows in physical or verbal outbursts, destructive behavior, abusive behavior, delusions, and even hallucinations.
Among the behavioral and psychiatric symptoms of Alzheimer’s, paranoia is one of the most disturbing. Behavior that’s characterized as paranoid and suspicious (particularly of others in cases where the patient has the aid of a caregiver or lives in a facility) may spur from feelings that they are losing control.
Patients with Alzheimer’s can become so delusional and suspicious of those around them that they become accusatory and charge caregivers, doctors, and co-residents of theft, infidelity, and lying. As a caregiver or relative, these accusations can be extremely hurtful, which is why it’s vital to keep in mind that the disease is causing this action.
Difficulty with planning and organizing is a common early warning sign of dementia. For instance, an elderly person may suddenly have difficulty planning their grocery shopping or finding their glasses that they always put in the same location. Although the exact cause of Alzheimer’s isn’t totally clear, the disease damages and kills brain cells, leading to fewer cells and fewer healthy brain connections among surviving cells.
The deterioration of brain cells causes significant brain shrinkage in the patient that can only be determined after death, during an autopsy. However, plaque (or clumps of protein forms on the brain) causes the destruction brain cells by disrupting cell-to-cell communication, and tangles that disrupt nutrient transport to healthy brain tissues. Alzheimer’s disease is also suspected to develop due to a variety enviromental, genetic, and lifestyle factors that ultimately affect the brain.
Agitation may be triggered by a variety of things, for instance environmental factors, frustration over the inability to carry out simple tasks, the inability to communicate clearly, fatigue, or fear as “control” (i.e., driver’s license, home) is suddenly taken away. Those stricken with Alzheimer’s, are usually the first to notice that there’s an issue.
However, it will be gradual. You may first notice you have difficulty remembering dates, times, details, and thoughts. However, when you notice that these changes are also noticeable by friends, coworkers, and family members, it can lead to embarrassment, anger, and agitation, and eventually lashing out and social withdrawal.
Hallucinations or delusionary thoughts are often experienced by dementia patients, most commonly visual (seeing things that aren’t there) or auditory (hearing noises that aren’t there)—such as the belief that a caregiver is out to get them without reason. Keep in mind that those with Alzheimer’s can experience both hallucinations and delusions.
A delusion is characterized as in the believing of false things (i.e., people, memories, details, and events). However, hallucinations are different. When suffering a hallucination, an individual has a false perception. This can be a false perception of an event, objects, or person that’s sensory in nature, meaning patients with Alzheimer’s can actually feel, hear, see, taste, and smell things that don’t really exist.
It’s common for Dementia patients to suddenly become sexual without awareness that their actions are inappropriate—for instance, removing clothing, exposing oneself in public, or touching and saying tasteless things to strangers and caregivers. This inappropriate sexual behavior can be embarrassing and even frightening for loved ones and caregivers.
These inappropriate and often aggressive sexual behaviors are part of the disease. For instance, a patient may become sexually dis inhibited (or bold) as damage becomes more severe in the brain’s frontal and temporal lobes, which is the area that manages control response.
This could be as simple as an inability to reason or a more drastic decline in cognitive functions—such as thinking, learning, reading and retaining information, problem solving, language and speech. Clinical researchers at the New York University School of Medicine’s Silberstein Aging and Dementia Research Center, point out that not all individuals experience the same rate of cognitive decline. For instance, many in the early stages of dementia show no memory loss or decrease in decision making abilities.
However, cognitive decline can enter in the mid- and later stages of the disease, resulting in trouble with memory, performance of mundane daily tasks, losing objects, the inability to use the right word, forgetting names or people, and trouble with planning, remember dates, and organizational skills.
Alzheimer’s is the most common cause of dementia, a general term for memory loss and other cognitive abilities serious enough to interfere with daily life. Alzheimer’s disease accounts for 60-80% of dementia cases.
Alzheimer’s worsens over time. Alzheimer’s is a progressive disease, where dementia symptoms gradually worsen over a number of years.
During this stage, Alzheimer’s disease is not detectable and no memory problems or other symptoms of dementia are evident.
The senior may notice minor memory problems or lose things around the house, although not to the point where the memory loss can easily be distinguished from normal age related memory loss. The person will still do well on memory tests and the disease is unlikely to be detected by physicians or loved ones.
At this stage, the friends and family members of the senior may begin to notice memory and cognitive problems. Performance on memory and cognitive tests are affected and physicians will be able to detect impaired cognitive function.
Patients in stage 3 will have difficulty in many areas including:
People with stage three Alzheimer’s may also frequently lose personal possessions, including valuables.
Personality changes that may include the opposite manners or personal
In stage four of Alzheimer’s disease clear cut symptoms of Alzheimer’s disease are apparent. Patients with stage four Alzheimer’s disease:
During the fifth stage of Alzheimer’s, patients begin to need help with many day to day activities. People in stage five of the disease may experience:
On the other hand, patients in stage five maintain a modicum of functionality. They typically can still bathe and toilet independently. They also usually still know their family members and some detail about their personal histories, especially their childhood and youth.
Patients with the sixth stage of Alzheimer’s disease need constant supervision and frequently require professional care. Symptoms include:
Stage seven is the final stage of Alzheimer’s disease. Because Alzheimer’s disease is a terminal illness, patients in stage seven are nearing death. In stage seven of the disease, patients lose ability to respond to their environment or communicate. While they may still be able to utter words and phrases, they have no insight into their condition and need assistance with all activities of daily living. In the final stages of the illness, patients may lose their ability to swallow.
Researchers pioneer technique to test how changes in brain activity may produce autism symptoms. Researchers altered the social behavior of mice by using light to manipulate electrical activity in a brain region involved in learning and socializing. The study, published this fall in Nature, bolsters the theory that autism may stem from an imbalance in the natural signals that excite or dampen activity within the brain. The study also offers a new approach to creating animal models of autism—crucial for testing promising medicines that might relieve disabling symptoms.
Using a technique, he pioneered and dubbed “optogenetics,” Stanford University psychiatrist Karl Deisseroth, M.D., Ph.D., and his colleagues engineered mice to produce light-sensitive proteins in the prefrontal cortex—a region involved in learning and social behavior. In a typical brain, some cells send signals that excite brain activity while other cells send signals that quiet it. In the optogenetic mice, excitatory brain cells respond to blue light and inhibitory brain cells respond to yellow light.
As a result, the researchers could dial up or dial down the level of activity in a mouse’s prefrontal cortex with pulses of light sent through a fiber optic cable implanted in its brain. The light’s effect lasted up to a half hour, enabling the researchers to remove the visible portion of the fiber optic implant and observe how the mice interacted with new mice or objects placed in their enclosures.
When the mice were exposed to blue light alone, they abruptly lost interest in socializing with new mice. By contrast, typical mice readily approach and sniff newcomers. However, the blue-light stimulated mice did not display other deficits such as difficulty adjusting to new objects placed in their cages.
When both excitatory and inhibitory cells were turned on simultaneously (by exposure to blue and yellow light), the mice resumed typical social behaviors.
The findings support a theory that autism stems from a dysregulation of normal brain signaling. Other evidence supporting this idea includes the fact that about one-third of those with autism also suffer seizures, a result of excessive electrical activity in the brain. In addition, several of the altered genes associated with autism play a role in brain signaling. Also, brain imaging studies reveal that some people affected by autism show higher than normal activity in brain regions associated with social behavior.
This latest experimental evidence further suggests that restoring balance to brain activity may be a way to relieve some of autism’s core symptoms. It also provides groundwork for future research investigating the role that specific brain circuits play in autism. Deisseroth and his colleagues are already developing new mouse models that will allow scientists to manipulate the activity of other brain regions and circuits, promising a more precise picture of how brain signaling problems might give rise to autism’s core symptoms.
Yizhar O, Fenno LE, Prigge M, et al. Neocortical excitation/inhibition balance in information processing and social dysfunction. Nature. 2011 Jul 27;477(7363):171-8.
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